Arch Gen Psychiatry. Subject race included Significant differences were observed in all 3 domains. Multivariate analysis using logistic regression identified the following 3 key predictors as being greater in the substance-induced group: parental substance abuse odds ratio [OR], 1. The key predictor of total positive and negative symptom score was greater in the primary psychosis group OR, 0.
If you already have a mental illness or it is diagnosed during treatment and assessment, particularly if psychosis is a reported symptom, further substance abuse can worsen symptoms when attempting to use alcohol or drugs as a psychsois of self-medication. However, there is evidence that caffeine, in extreme Artificialky doses or when severely abused for long periods of time, may induce psychosis. People who take drugs but do not become dependent on them are mpdel far less Artificially induced model psychosis for psychosis, which means that in the majority of cases those Investments pants drug-induced psychosis will Artificially induced model psychosis substance use disorder treatment in addition to mental health services. Neuropsychopharmacology 29, — Can Marijuana Cause Psychosis? Hallucinogens like LSD and PCP cause altered perceptions and distortions of reality, and in some instances heavy, regular users may not be able to escape these surreal states.
Teen group blowjobs. What is Drug-Induced Psychosis?
Discussion Our patient experienced a first-episode of psychosis related to use of SCs, with disorganized and paranoid thought processes. This model also provides a rationale for future studies of preventive anti-psychotic treatment, including psycho-social, psycho-educational and possibly also pharmacological interventions to decrease vulnerability, in the same way as is recommended for individuals with a primary psychotic disorder. June Artificially induced model psychosis Rev Artificially induced model psychosis. Frequency of use may be influenced by multiple factors including a desire to get high while avoiding a positive urine toxicology, the curiosity for trying a new drug, and Aritficially misconception that it is not illegal and therefore it is safe [ 12 ]. Beware of ex-KGB agents who are cooperating with the U. Finally, even if you are caught red-handed, there must Artificialpy a trial before imprisonment. South Australia: Drug Teacher bates donna Alcohol Services; J Neuroimmune Pharmacol. The Warren Comission found him indufed credible. References 1. Methamphetamine abuse: a perfect storm moeel complications. Look this term up in a medical dictionary. A rating scale for evaluation of the clinical course and symptomatology in amphetamine psychosis.
In the early part of the 20th century, modern-day illicit drugs were once used to treat a variety of mental illnesses and ailments.
- Use of amphetamine and methamphetamine is widespread in the general population and common among patients with psychiatric disorders.
- Look this term up in a medical dictionary.
- After an interruption of almost 20 years, psychopharmacological research on hallucinogenic drugs was revived in several countries simultaneously around
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- At American Addiction Centers, we strive to provide the most up-to-date and accurate medical information on the web so our readers can make informed decisions about their healthcare.
- Synthetic cannabinoids- SCs- induced psychosis is a growing public health concern.
Use of amphetamine and methamphetamine is widespread in the general population and common among patients with psychiatric disorders. Amphetamines may induce symptoms of psychosis very similar to those of acute schizophrenia spectrum psychosis. This has been an argument for using amphetamine-induced psychosis as a model for primary psychotic disorders. To distinguish the two types of psychosis on the basis of acute symptoms is difficult.
However, acute psychosis induced by amphetamines seems to have a faster recovery and appears to resolve more completely compared to schizophrenic psychosis. The increased vulnerability for acute amphetamine induced psychosis seen among those with schizophrenia, schizotypal personality and, to a certain degree other psychiatric disorders, is also shared by non-psychiatric individuals who previously have experienced amphetamine-induced psychosis.
Schizophrenia spectrum disorder and amphetamine-induced psychosis are further linked together by the finding of several susceptibility genes common to both conditions. These genes probably lower the threshold for becoming psychotic and increase the risk for a poorer clinical course of the disease. The complex relationship between amphetamine use and psychosis has received much attention but is still not adequately explored.
Our paper reviews the literature in this field and proposes a stress-vulnerability model for understanding the relationship between amphetamine use and psychosis. Amphetamine and methamphetamine hereafter amphetamines can prolong wakefulness, increase focus and feelings of energy as well as decrease fatigue.
Adverse effects include anxiety, aggression, paranoia, hyperactivity, reduced appetite, tachycardia, increased breathing rate, dilated pupils, increased blood pressure, headache, insomnia, palpitations, arrhythmia and others [ 1 ]. Amphetamines inhibit dopamine reuptake by interacting with the dopamine transporter DAT , thereby increasing the concentration of dopamine in the synaptic cleft [ 2 ].
Amphetamines also interact with the vesicular monoamine transporter 2 VMAT2 , leading to increasing amounts of dopamine in the cytosol, a possible mechanism of action for the neurotoxicity of amphetamines. Neurotoxic effects are seen also in serotonergic and noradrenergic neurons. Amphetamines are highly addictive drugs.
Abuse of amphetamines is widespread in the general population [ 4 - 9 ]. It is also common among psychiatric patients [ 10 , 11 ], where a high percentage test positive for amphetamines [ 12 ]. There is overwhelming evidence that patients with psychotic disorders have an increased vulnerability to compulsive use of drugs of abuse [ 13 , 14 ], including psycho stimulants like amphetamines [ 15 ].
This may be especially true for patients with schizophrenia like disorders [ 16 ]. There may be several explanations for this increased co morbidity, but there is convincing evidence from animal studies that this may be due to shared vulnerabilities for both psychosis and drug use disorders [ 17 ].
These animal studies also point to possible neural mechanisms explaning the increased co morbidity [ 18 ]. The association between amphetamine use and psychosis has received much attention [ 19 ], but several questions about this complex relationship remain unanswered. In the following, we review some of these questions and propose a new model for understanding the relationship between amphetamine use and psychosis.
Observations strongly suggest a relationship between the intake of amphetamines and the development of acute psychosis. First, early studies demonstrated that amphetamines could trigger acute psychosis in healthy subjects. In these studies, amphetamine was given in consecutively higher doses until psychosis was precipitated, often after — mg of amphetamine [ 20 - 23 ].
The symptoms subsided within 6 days. The effect was blocked by the use of anti-psychotics [ 24 ]. Not all the subjects in these studies became psychotic, as some had to be removed from the experiment because of health risks caused by elevation of heart rate, blood pressure or body temperature. Secondly, psychosis has been viewed as an adverse event, although rare, in children with ADHD who have been treated with amphetamine [ 25 - 30 ].
The wide variation is probably due to different populations being studied, gender [ 38 ] and the method and duration of amphetamine use [ 39 ]. It may also depend on the instruments used to assess psychosis, e. Lastly, there is a positive correlation between amphetamine availability at a community level and the incidence of psychosis in the same population [ 41 - 44 ].
There is clinical evidence that such binges may end in psychosis. Surprisingly, it is poorly understood [ 1 , 50 ], whether such psychosis is due to amphetamine use per se amount over time, amount on one occasion or the length of the current binge , vulnerabilities in the user or both.
It could be that psychosis occurs because of the sleep deprivation that follows amphetamine use, or because of other factors at the end of a binge. Users will often end their binge by using sedating drugs like alcohol, benzodiazepines, opiates or cannabis. This could be viewed as self-medication [ 51 ] and may be one reason why users often develop problems with several drugs. Only a weak relationship has been reported between psychotic symptoms and the level of amphetamines in the blood of psychotic patients [ 51 , 52 ].
This could be because acute blood levels at the end of a binge are a poor representation of overall amphetamine exposure, but it could also be because individual vulnerability, rather than level of amphetamine exposure, is the critical risk factor for developing acute psychosis. The symptoms of psychosis induced by amphetamines are very similar to those of acute schizophrenia spectrum psychosis and include: lack of concentration, delusions of persecution, increased motor activity, disorganization of thoughts, lack of insight, anxiety, suspicion and auditory hallucinations [ 21 , 22 , 53 ].
Some studies have suggested differences with more pronounced grandiosity and visual hallucinations [ 52 , 54 ]. The thought disorders that occur in schizophrenia characterized by a splitting and loosening of associations, a concreteness of abstract thought, and an impairment in goal-directed thought, may be less prominent in amphetamine induced psychosis [ 55 ]. However, distinguishing the two types of psychosis on the basis of acute symptoms is probably very difficult [ 56 ].
The similarities between the two conditions are, in fact, so pronounced that this has been used as an argument for using amphetamine-induced psychosis as a model for primary psychotic disorders [ 21 , 54 , 57 - 59 ].
In contrast to schizophrenic psychosis, acute psychosis induced by amphetamines seems to have a faster recovery [ 60 - 63 ], and appears to resolve with abstinence, although the recovery may be incomplete [ 43 ]. Japanese researchers have argued that psychosis induced by amphetamines could, in fact, be of much longer duration, up to several years [ 64 - 66 ].
Stressful situations seem to trigger such flashbacks in susceptible individuals and several vulnerability factors have been identified, e. It is difficult to distinguish the Japanese chronic amphetamine psychosis from a primary psychosis triggered by the use of amphetamines [ 64 ], although it has been claimed that they constitute separate entities. In animal models, there is sensitization to the rewarding effects of amphetamines e.
Sensitization is also seen in human subjects [ 72 ]. There is reason to believe that an earlier psychosis involves a risk of future psychotic episodes due to this sensitization [ 43 , 73 - 75 ], or possibly to the development of dopaminergic super sensitivity [ 76 , 77 ].
Psychosis may be precipitated acutely by amphetamine due to its effects on dopaminergic activity in the CNS [ 46 ]. In the longer term, the neurotoxic effects of the drugs on serotonin and dopamine neurons [ 78 ] and dopamine transporters [ 79 ] may play a role.
Amphetamine sensitization seems to cause dysregulation of dopamine by the ventral subiculum [ 80 ]. There is an over-expression of the dopamine receptor, subtype 2 DRD2 [ 81 ] and a higher sensitivity of DRD2 to the effects of amphetamines in vulnerable individuals [ 82 ]. In addition to the increased risk of psychosis following the use of amphetamines in people who have experienced amphetamine-induced psychosis previously, patients with schizophrenia [ 83 ] and schizotypal personality traits [ 74 , 84 ] may more readily become psychotic after the use of amphetamines.
Other risk factors for psychosis may include amphetamine use disorders abuse and dependence , the presence of other psychiatric disorders primarily attenuated psychosis syndrome, personality disorders and affective disorders , early cognitive dysfunction such as those found in the prodromal states of schizophrenia , family history of mental disorder and the use of other drugs like opiates, benzodiazepines, cannabis and alcohol [ 37 , 67 , 74 , 75 , 85 - 87 ]. Several susceptibility genes have been found in common for amphetamine-induced psychosis and schizophrenia [ 32 , 58 ].
These genes increase the risk both for becoming psychotic and for a poorer clinical course of the disease. Studies in Japan also indicate that primary and drug induced psychosis may be genetically linked. Relatives of methamphetamine-users with a lifetime history of amphetamine psychosis are 5 times more likely to have schizophrenia than methamphetamine-users without a history of psychosis [ 85 ].
Patients with schizophrenia and those with psychosis induced by amphetamines both show significantly increased peripheral plasma levels of norepinephrine compared to methamphetamine users who do not have psychosis, and to non-using conrols [ 65 , 88 ]. This seemingly common vulnerability is important considering the difficulties in distinguishing between the two conditions in the long term. The precipitation of psychosis by amphetamines in healthy subjects can be blocked by anti-psychotics [ 24 , 89 ].
Similarly, psychotic symptoms caused by amphetamines can, like acute schizophrenic psychosis, be treated with anti-psychotics [ 90 ]. A Cochrane review from [ 91 ] identified only one randomized controlled trial of treatment for psychosis induced by amphetamines which met the criteria for included studies.
It showed that both olanzapine and haloperidol in clinically relevant doses were effective in treating psychotic symptoms [ 92 ]. One problem with using anti-psychotics could be that such drugs have a tendency to block the DRD2, potentially increasing anhedonia that could, in turn, cause a greater vulnerability to relapse into drug abuse. Some studies indeed point in this direction [ 93 - 95 ]. The use of alternative therapeutic drugs, such as benzodiazepines, will reduce the chance of extra pyramidal adverse effects [ 96 ] and decrease the risk of intoxication [ 97 ].
However, one argument against this strategy is that anti-psychotics seem to protect against the neurotoxic effects of amphetamines [ 98 , 99 ].
This leaves us with a dilemma - how valid is a diagnostic construct of amphetamine induced psychosis? Such a term suggests the assumption that this type of psychosis can be induced in individuals otherwise not susceptible to, e. These researchers identified 18 papers that specifically focused on delineating the clinical characteristics or outcomes of individuals diagnosed with substance-induced psychosis.
Seven of these papers focused on stimulants amphetamines and cocaine , but only one had a 1 year follow-up assessment. Also for other substances of abuse with a tendency to cause psychosis there have been similar discussions. It has long been recognized that the use of cannabis in early adolescence increases the risk of later development of psychosis and schizophrenia [ , ]. Because the drug intake takes place many years before the diagnosis of schizophrenia it has been argued that this cannot be a case of reversed causality [ ].
There are however some arguments in the opposite direction. Firstly, even with a formidable increase in the use of cannabis in the population, no increase in the incidence of schizophrenia has been observed [ ].
Secondly, it has been shown that patients with schizophrenia may have their psychosis triggered by lower intake of cannabis than healthy volunteers [ ].
Lastly, we do not know when a psychotic disorder starts to develop. It may be that it starts long before the initial psychotic symptoms, opening for the possibility of reversed causality, even when intake of cannabis takes place years before first psychotic episode [ ].
The present agreement in the field seems to be that cannabis can precipitate psychosis in vulnerable individuals, an agreement that closely resembles our suggested model. The similarities between acute schizophrenic psychosis and psychosis following the use of amphetamines are so pronounced that the latter has been suggested as a model for schizophrenia [ 58 , ]. However, it remains unresolved whether the relationship between amphetamines and psychosis is explained by drug exposure amphetamine-induced psychosis , amphetamines use triggering a primary psychotic disorder or both.
Although psychosis may be induced by amphetamine in healthy individuals, not all subjects become psychotic by the doses of amphetamines allowed in the experiments. Some, but not all, individuals using amphetamines have experienced psychotic episodes, and a few have experienced psychosis as an adverse event during stimulant treatment.
Is this a result of differences in amphetamine exposure or differences in vulnerability? Furthermore, psychosis is precipitated by a lower dose of amphetamines in individuals with primary psychosis and may be blocked by the use of anti-psychotics.
Finally, there seem to be many genetic and physiological similarities between amphetamine-induced psychosis and acute schizophrenic psychosis, suggesting that vulnerability may play a significant role in the occurrence of amphetamines-induced psychosis. Exposure to amphetamines should be viewed as a stressor in the acute phase for the vulnerable individual in a dynamic way; for individuals with lower vulnerability higher doses of amphetamines are needed, whereas individuals with higher vulnerability require lower doses to precipitate acute psychosis.
In addition, due to their sensitizing effects, amphetamines may also play a role in the development of vulnerability. Repeated use of amphetamines could increase vulnerability, thereby increasing the chances of developing psychotic symptoms even in the absence of acute exposure to amphetamines. Thus, primary psychotic disorder and psychosis precipitated by amphetamines need not be considered as two separate phenomena, but as two phenomena interlinked in a dynamic way.
Some users of amphetamine will not develop psychosis even using high amounts, while others will develop psychosis with little or no exposure. Taking such a view may have some important clinical implications.
A reported alcohol consumption times per week, with an average of mixed alcoholic beverages on each use, in social settings. Due to the end of the Cold War, numerous Eastern technicians who have conducted similar research for Eastern European governments during the Cold War are now also flooding into the U. Clinical features of sensitization to methamphetamine observed in patients with methamphetamine dependence and psychosis. The second major type of CB, CB2 receptors, is mainly found in the peripheral nervous system, in the immune system where they mediate the immunomodulatory effects of cannabinoids, and only sparsely in the central nervous system [ 2 ]. As the autopsy of Robert F. Look this term up in a medical dictionary.
Artificially induced model psychosis. What is drug-induced psychosis
Studies such as these underscore the connection between substance abuse and psychosis. Substance intoxication or withdrawal may bring on psychotic symptoms, and when this occurs, crisis intervention methods and medical detox may be necessary.
It can be helpful to understand how substance abuse and psychosis are intertwined, and which one may have predated the other, when determining treatment needs. When psychosis or mental illness occurs first, patterns of substance abuse may develop later a means of self-medication. In such an instance, treatment protocols may focus on not only managing a combination of acute intoxication and psychotic symptoms, but instituting a longer-term strategy—including behavioral therapeutic interventions and psychiatric medications—to manage the underlying mental health issue.
If the substance abuse occurred first and the psychosis is a byproduct of drug use, the focus will be on managing the acute but transient psychotic symptoms with antipsychotics or anxiolytics.
Sometimes, just a quiet and safe place is all that is needed. Once symptoms resolve, standard addiction treatment can begin. Certain drugs are associated with symptoms that could resemble certain mental illnesses. See below. Withdrawal from depressants may also bring about mental health symptoms such as anxiety, perceptual disturbances, hallucinations and other perceptual disturbances, or delirium.
In some cases, long-term substance use may also be associated with depression and suicidal thoughts. In heavy meth users, psychotic symptoms may last months or even years past the point of quitting.
In many cases, psychosis may go away after the substance abuse is stopped; however, in others, symptoms may persist well beyond the point of substance use.
A severe psychotic episode may require may require hospitalization to stabilize an individual both mentally and physically in a safe and secure environment that can provide medical and mental health monitoring and care. Drug-induced psychosis may seem self-explanatory as to the cause—psychotic symptoms are brought on by drug abuse.
Drugs can affect different people in a variety of ways, and what may cause psychotic symptoms in one person may not in another. Additionally, it is not only that drug use can contribute to the development of mental health issues, but also that mental health disorders can contribute to substance use. While in some cases, it may appear that mental health symptoms sprung from the drug use, what actually might be occurring is that untreated symptoms of an underlying mental health condition were exacerbated by the substance abuse but not caused by it.
Further, both mental illness and addiction have certain overlapping risk factors, so some individuals may be more likely to struggle with both addiction and mental illness.
Certain drugs sensitize neurons in the brain, and this sensitization may lead the person to use those drugs more frequently and in higher amounts over time. Mental health disorders often follow a similar pattern of increasing severity, where episodes that originally occurred only occasionally will begin to happen more and more often with increasingly brief periods of respite between. For those who are genetically vulnerable to such a kindling phenomenon, both addiction and mood disorders may be more likely to develop.
Co-occurring psychotic disorders and addiction can worsen the symptoms of both issues. Such a subliminal device can be operated in both the Rf as well as in the microwave frequencies.
Another way is to hook up a Neurophone to a police-type audio doppler radar transmitter, transmitting in the K-band police radar frequencies. There are of course numerous other subliminal techniques. In order to obtain a more detailed technical understanding of these its often very helpful to check out the various "HEARING AID"-patents for the deaf as the military and classified "black" research community has done the same to turn these into subliminal buck rogers devices and weapons. Another way to obtain more detailed technical information is to discretly interview the same scientists and electronical engineers who have done this research for the Intelligence Agencies.
You will come across their names if you search the homepage of the U. Patent and Trademark Office and use the Bolean search system supplied there to search the cover pages of the patents of interest.
Due to the end of the Cold War, numerous Eastern technicians who have conducted similar research for Eastern European governments during the Cold War are now also flooding into the U.
Its often extremely helpful to talk to them. Beware of ex-KGB agents who are cooperating with the U. It has been fairly well documented that Chapman was psychotic when she shot Lennon. Before the assassination, in the advanced stages of his model psychosis, Chapman sat in his room in Hawai and kept chanting over and over and over :. What we can now deduct here is that the government has secretly continued to develop this highly classified technique to eliminate all human contact between the assassin and his "inducer".
This is the important distinction here : I am not proposing that subliminal technologies exist which can make someone assassinate a preselected target even if the person it is suggested to is suffering from the advanced stages of an artificially induced model psychosis.
Rather, what I am saying here is that the subliminal messages are used to trick the Manchurian-Candidate-in-the- Making" to psych himself up. And once in the grips of a model psychosis, he will not be able to grasp the significance of the messages he is chanting or writing. It was the FBI of course who because of the contrast between his personal wealth and his involvement in social and socialist causes had long considered John Lennon to be a phony. Why would the government want to assassinate John Lennon.
Remember this was the time of the Cold War. Ronald Reagan had just been elected for the promise to "make America strong again". This entailed building up the military, heating up the arms race and engage in numerous insurgencies in Third World countries. The government didn't need a John Lennon capable of motivating millions of people to march in "Give Peace A Chance" demonstrations.
They were afraid of the political impact he would have. Newly released documents prove that during the last days of WWII, the British government considered several plots to assassinate Adolf Hitler.
One of the plots considered was to hypnotize Rudolf Hess to shoot Hitler. As we have meticulously discussed, hypnosis is useless for this purpose. The point I am trying to make is that if the British government experimented with these techniques back in the early 's, they surely must be aware that Mark Chapman is the victim of an artificially induced model psychosis.
Do not confuse a psychopath like Ted Bundy, for example who has no conscience about the crimes he commits with someone who committs a crime because he is suffering from a psychosis so that he is hallucinating and can thus no longer distinguish between right and wrong.
A psychopath, on the other hand, knows the difference between right and wrong. He is not hallucinating and understands that his actions constitute a crime. He simply doesn't have a conscience. Chapman committed the crime as a result of a psychosis which had progessed to the point where he was hallucinating and not in touch with reality anymore. The question NEVER EVER addressed by the mainstream investigative newsmedia is whether Chapman's psychosis was the result of a genuine "internal" mental imbalance or an artificially induced model psychosis which is the result of "external" manipulation.
Unfortunately, even a highly trained, experienced psychiatrist will not be able to distinguish an artificially induced "external" model psychosis from a genuine "internal" psychosis which develops due to mental illness. With regards to the assassination of Robert F. Kennedy's brother : The autopsy of RFK's body clearly proves beyond any reasonable doubt that RFK was shot from behind by a gun which due to the burn marks it left on his clothing could not have been more than a few inches from his body.
The autopsy was performed by Dr. Thomas Noguchi I'm recalling his name from memory and may be spelling it wrong.
Sirhan also held his arm with the gun at a straight angle while RFK's autopsy proves his killer shot upwards so that the bullets which really killed RFK ended up stuck in the ceiling tiles which the LAPD promptly removed and subsequently conveniently trashed. It is easy to verify by discussing this technique with knowleable psychiatrists, for example that an individual suffering from the advanced stages of an artificially induced model psychosis can easily be manipulated to commit acts of violence he or she would not normally commit.
Another item of interest that should be highlighted here is that moments before Chapman assassinated John Lennon, the Dakota's nightwatchman, Jose Perdomo, a Cuban exile, was discussing the assassination of John F. Kennedy with Mark Chapman.
See article s in the weekly People Magazine by James R. Gaines, sometime in the 's Mark Chapman later said, "That assassination has always meant a great deal to me! Remember that Jack Ruby was being psyched up by Dallas police officer Olson, that "Lee Harvey Oswald should be cut inch by inch into ribbons", before he shot him. Ever since Watergate, it is no longer a national secret that many Cuban exiles are closely associated with the Central Intelligence Agency.
Jose Perdomo's intelligence associations should be immediately investigated!!! The literary story and content of J. Salinger's book "The Catcher in the Rye" has absolutely no connection to the asassination of John Lennon.
Salinger's story. In order to brainwash an innocent, unwitting individual via an artificially induced model psychosis to hallucinate that he must carry out the asassination of a preselected target it is necessary to trick the "Manchurian-Candidate-In-the-Making" to psych himself up.
For this purpose it is very helpful to subliminally induce the subject to repeatedly chant or write down a rhym connected with the subliminal assassination message i. In addition, it is quite plausible that the term "Catcher In The Rye" denotes a secret clique of Intelligence officials FBI, CIA, etcetera who for obvious reasons can't bragg openly about how they pulled off the assassination of John Lennon.
And we all know what happens at any regular university where large numbers of people come together : They soon form cliques. This is even more true in the world of intelligence and covert operations. If you are interested in their mindcontrol technologies a great place to start a technological search can be found at :.
To build a device which broadcasts audio into the human head via microwaves, connect a police type doppler radar transmitter to the Experimenter's Neurophone listed on Terry Bastian Neurophone homepage see above , then test all frequencies by hooking it all up to a Sweep Oscillator be sure to check out the GHZ range, but don't leave it at that.
As you may know, its a rather conservative book which asserts that this mind control stuff was carried out but didn't work. And Chapman was indeed suffering from a psychosis when he shot John Lennon. The real trick is to trick the "Manchurian-Candidate-In-The-Making" to psych himself up once the artificially induced model psachosis has progressed within the individual to a certain stage i.
In Sirhan Sirhan's case this was accomplished by manipulating him into practicing the rituals of the Rosecrucians. These were to sit in front of his mirror and write his thoughts down into his notebook. Once this initially harmless exercise became a habbit for Sirhan, his model psychosis was clandestinely induced.
There are in fact two ways in which this can be accomplished. The easiest is via Methamphetamine overintoxication -- and it only takes five to seven days of continuing drugging to accomplish that. A more "modern" way to induce a psychosis is to subject the subject to microwaves in the 2. One such a device, which I am currently in the process of putting together utilizes a so-called audio-band pink noise generator which generates a low-frequency white noise signal.
This low-frequency white noise signal is modulated on a high-frequency generator, sent over a Haan high-frequency gunn diode MA gunn diode does the job and exits via an antenna Feedhorn Assy. Microwave induced stress is cumulative and it may take several weeks to induce the psychosis where the individual is at the stage that he can no longer resist the subliminal and psychological trickery which is then necessary to induce him to commit the assassination.
Back to Sirhan : Once he had reached the "desired" stage of the psychosis, the "thoughts" which came to his mind while sitting in front of his mirror are clearly reflected in what -- by continuing to carry out the practices of the Rosecrucians -- he wrote into his notebook, i.
All I'm saying here is that due to the other evidence of the crime scene, i. Kennedy was shot from behind while Sirhan stood in front of him as the autopsy proves I believe that these "thoughts" were not Sirhan's own but rather subliminally induced.
Just like any other technologies, the technique was clandestinely improved upon. Just like writing the assassination message into his notebook, chanting it repeatedly also tricks a subject suffering from the advanced stages of an artificially induced model psychosis into psyching himself up. We should also keep in mind here that such subliminal devices need not be bigger than a small ghetto-blaster, stereo-walkman or pocket radio to which an individual in an advanced psychotic state of mind will exhibit a much greater reaction than a person in a "normal" state of mind.
These devices can be clandestinely hidden in the objects of his or her surroundings i. So, now that I have spilled the beans for all you curious internet browsers, aren't you curious how I figured it all out??? Gaines in the weekly People Magazine, June 22nd, Gaines, February Part I of a three-part series.
God changed something in my heart and He spoke to my heart so that I could hear Him through all the sickness that was in my mind at that time. I was out of my mind and I wanted to go to that trial worse than anything in the world. So why would I plead guilty and just walk away from the best chance I would ever have to promote the book that had become my life [ My lawyer didn't want me to plead guilty.
The psychiatrists didn't want me to plead guilty, and they tried to stop me from doing it. I wasn't listening anymore. The voice of God? Excuse me, but we know better, don't we, Mr. So there you have it. Read the book called "Who Shot John Lennon? The author asks a single question I've never heard anyone in the world ask, and it puts the whole case against the FBI to bed!
Simpson's trial! Don't you think it would have been even bigger over John Lennon? Finally, even if you are caught red-handed, there must be a trial before imprisonment.
Substance-induced psychosis - Wikipedia
Psychosis is a mental health problem which temporarily causes someone to interpret the world differently from those around them. Psychosis is often characterised by delusions or hallucinations, which are experiences that are far removed from reality.
Delusions are irrational beliefs that a person holds, even when they are presented with evidence that contradicts these beliefs. Delusions may include believing that you have a serious or life-threatening physical illness, that you are responsible for terrible things happening to other people, or that you are bankrupt when you are not.
Hallucinations refer to intense sensory perceptions of phenomena that are not real, and are characterised by individuals vividly feeling, seeing or hearing things that do not truly exist. Drugs such as cocaine , cannabis and hallucinogens can worsen symptoms of existing mental illnesses, whilst taking such substances for a long period of time can also see you develop symptoms of psychosis, including paranoia. If you are diagnosed with drug-induced psychosis and have a dependency on alcohol or drugs , you may have what is called a dual diagnosis, whereby your underlying mental illness will need to be treated independently of any substance addiction.
At Priory, our nationwide network of hospitals and wellbeing centres provide highly flexible, specialist treatment to treat both underlying mental health disorders and drug or alcohol abuse across two separate treatment plans, created specifically with your needs in mind.
Treatment for underlying psychiatric disorders such as bipolar disorder and schizophrenia will involve a combination of talking therapies and medication designed to reduce symptoms, while substance abuse treatment can include withdrawal and detox plans within a safe and understanding environment.
Drug-induced psychosis can happen when you take too much of a certain drug, so that its level of toxicity provokes paranoia and a psychotic episode. It can also occur when if you have an adverse reaction from mixing different substances, or withdrawing from a drug, prescribed or otherwise.
You may already have underlying mental health conditions such as bipolar disorder or schizophrenia, where delusions and hallucinations are associated symptoms, with psychotic episodes resulting from substance use, indicating that you may be prone to psychosis. If your psychosis has been triggered through drug use in order to cope with underlying mental illness, a diagnosis would need to determine whether symptoms would continue without the drug present, as this would not be drug-related psychosis.
Drug-induced psychosis is more apparent when your symptoms wear off after you have stopped using the drug, while the initial symptoms such as social withdrawal and lack of motivation may gradually build to include delusions or hallucinations.
If you are diagnosed with drug-induced psychosis, it is vitally important that you seek treatment from specialist medical professionals. When treating drug-induced psychosis, the first step towards recovery must include termination of use of the drug which initially caused the episode. The medically assisted withdrawal detoxification programme at Priory will also ensure that you have no further interaction with drugs that could be habit forming or that could interfere with the drug being abused, with carefully selected drugs helping with symptoms of withdrawal only.
During the detox process, you will also have access to therapeutic sessions where you will learn how to manage withdrawal symptoms, and also prepare techniques for remaining drug-free after treatment. Residential secondary care programmes are available after detox, which can further help your transition and acquire healthy habits to help support clean living.
While removal of the drug from your system may remove symptoms of psychosis, if you have an underlying mental health condition such as anxiety or depression which prompted excessive use of a drug, or you have also been diagnosed with an existing psychotic disorder such as bipolar disorder or schizophrenia, further treatment will be required to address these conditions.
Therapies and medication can be offered on an inpatient, outpatient or day care basis depending on your circumstances, and may include:. Cognitive behavioural therapy CBT - once you have been medically stabilised, the practical and problem-solving talking therapy, CBT, can help you to learn more about the thoughts and moods that you experience before psychotic episodes occur, helping you to manage your emotions and paranoia more readily, and be aware of triggers.
If a mental health condition such as anxiety or depression exists, which has been masked by extensive use of drugs or alcohol to the point of psychosis, then CBT can help you to learn techniques to cope with and reduce associated symptoms so as to prevent relapse.
Family therapy - due to the serious nature of drug-induced psychosis and associated psychotic conditions, family therapy involving those closest to you, can help to ensure that there is sufficient support available at home to prevent relapse and manage associated symptoms, potentially reducing the need for extensive hospital treatment.
Anti-psychotic medication — if your underlying mental health condition features psychotic episodes as a symptom, then antidepressant, anti-psychotic or other medications such as clozapine Clozaril may be recommended for an extended period of time, particularly if delusions and hallucinations are frequent or particularly severe.
You can read more about the different treatment options that we offer at Priory, by accessing our approach to addiction treatment and approach to mental health treatment pages. To view all Priory drug-induced psychosis specialists, please click here. This can either exacerbate or trigger the onset of mental illnesses such as bipolar disorder and schizophrenia, which can be characterised by symptoms of psychosis, due to being predisposed to the condition.
If you are diagnosed with drug-induced psychosis, it will often be part of a dual diagnosis of substance addiction and underlying mental illness, which will need to treated as separate issues to aid effective recovery or reduction of symptoms. This means that you will undergo a medically assisted withdrawal detoxification programme in order to become medically stable, before beginning an individual treatment plan for any underlying mental health issues which may have contributed to the onset of psychosis symptoms.
It can be difficult to diagnose drug-induced psychosis when a mental illness has previously been diagnosed or is underlying throughout substance abuse, as symptoms of psychosis need to be related directly to drug intoxication, as opposed to being part of the mental illness itself.
You may only be officially diagnosed with drug-induced psychosis after a detox programme, as if the symptoms continue after the drug is removed from your system, psychosis cannot be drug-induced, while an improvement in symptoms after the drug has worn off will point to a drug affected episode.
If you have experienced an episode of drug-induced psychosis, there is a greater risk of having further psychotic episodes in the future. If you already have a mental illness or it is diagnosed during treatment and assessment, particularly if psychosis is a reported symptom, further substance abuse can worsen symptoms when attempting to use alcohol or drugs as a form of self-medication.
The symptoms of drug-induced psychosis are often gradual, with toxicity of the drug becoming more dangerous as the frequency and dosage of the drug increases with dependency. If you have an underlying mental health condition, then use of psychoactive drugs will likely worsen your symptoms, result in extreme paranoia, and can speed up the onset of psychotic disorders such as bipolar disorder and schizophrenia.
The drugs which are often reported in cases of drug-induced psychosis, and are most likely to result in symptoms of psychosis, include:. The precise symptoms that the above drugs may cause during drug-induced psychosis, varies depending on the drug in question, although some of the most commonly reported symptoms of drug-induced psychosis include:.
Auditory hallucinations involve hearing voices, such as a voice narrating your movements or actions, or two separate voices arguing between each other. While it may seem clear that drug-induced psychosis will be caused by an addiction or negative withdrawal process from the drug in question, people struggling with drug-induced psychosis often want to understand the root cause of their difficulties.
The delicate balance of chemicals in the brain which regulate our emotions, moods and behaviours can be affected when taking psychoactive drugs, interfering with the structure and function of your brain. The specific diagnosis of drug-induced psychosis is vitally important for appropriate treatment, as the symptoms of psychosis could also relate to underlying mental health conditions and biological factors, or even as a result of a recent traumatic event.
If you are diagnosed with drug-induced psychosis, it will likely be from extensive use or withdrawal of psychoactive drugs ranging from cannabis to LSD, while cocaine use is commonly associated with symptoms of psychosis both in the short and long-term. It is also possible that diagnosis of mental health conditions and psychosis-related disorders such as bipolar disorder and schizophrenia earlier in life leads to over-reliance on drugs or alcohol in order to cope with associated symptoms, although this is often likely to make symptoms worse in the long-term.
For professionals looking to make a referral, please click here. Drug-Induced Psychosis. Explore this section. What is drug-induced psychosis? What are the signs and symptoms of drug-induced psychosis? What types of drugs can cause drug-induced psychosis? What causes drug-induced psychosis?
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